Fitches, A C <br/><br/>
Creation of an additional glycosylation site as a mechanism for type I antithrombin deficiency.  [electronic resource] 

 -  Thrombosis and haemostasis  Oct 2001 
 - 1023-7 p.  digital 
<br/><br/> Publication Type: Case Reports; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0340-6245 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adolescent<br/>Adult<br/>Amino Acid Substitution<br/>Animals<br/>Antithrombin III--biosynthesis<br/>Antithrombin III Deficiency--classification<br/>COS Cells<br/>Cell-Free System<br/>Chlorocebus aethiops<br/>Exocytosis<br/>Female<br/>Glycosylation<br/>Humans<br/>Male<br/>Molecular Weight<br/>Mutagenesis, Site-Directed<br/>Mutation, Missense<br/>Pedigree<br/>Point Mutation<br/>Protein Folding<br/>Protein Processing, Post-Translational<br/>Recombinant Fusion Proteins--biosynthesis<br/>Substrate Specificity<br/>Thrombophilia--genetics<br/>Transfection<br/>Venous Thrombosis--etiology