McKenna, I M <br/><br/>
Heterozygous inactivation of TGF-beta1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras.  [electronic resource] 

 -  Toxicology letters  Sep 2001 
 - 151-8 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0378-4274 
<br/><br/><label>Standard No.: </label>10.1016/s0378-4274(01)00393-9  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenoma--chemically induced<br/>Animals<br/>Carcinogenicity Tests<br/>Carcinogens--administration & dosage<br/>Carcinoma--chemically induced<br/>Crosses, Genetic<br/>DNA Mutational Analysis<br/>DNA, Neoplasm--chemistry<br/>Female<br/>Genes, ras--drug effects<br/>Genetic Predisposition to Disease<br/>Genotype<br/>Heterozygote<br/>Lung Neoplasms--chemically induced<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred Strains<br/>Mutagenicity Tests<br/>Mutation<br/>Polymerase Chain Reaction<br/>Polymorphism, Single-Stranded Conformational<br/>Transforming Growth Factor beta--deficiency<br/>Transforming Growth Factor beta1<br/>Urethane--administration & dosage