Sheng, J G <br/><br/>
Interleukin-1 promotion of MAPK-p38 overexpression in experimental animals and in Alzheimer's disease: potential significance for tau protein phosphorylation.  [electronic resource] 

 -  Neurochemistry international   
 - 341-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0197-0186 
<br/><br/><label>Standard No.: </label>10.1016/s0197-0186(01)00041-9  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Aged, 80 and over<br/>Alzheimer Disease--metabolism<br/>Animals<br/>Blotting, Western<br/>Brain--enzymology<br/>Female<br/>Humans<br/>Interleukin-1--genetics<br/>Male<br/>Middle Aged<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Phosphorylation<br/>RNA, Messenger--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>p38 Mitogen-Activated Protein Kinases<br/>tau Proteins--metabolism