Disruption of Sur2-containing K(ATP) channels enhances insulin-stimulated glucose uptake in skeletal muscle. [electronic resource]
- Proceedings of the National Academy of Sciences of the United States of America Sep 2001
- 11760-4 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0027-8424
10.1073/pnas.201390398 doi
ATP-Binding Cassette Transporters Analysis of Variance Animals Biological Transport Blood Glucose--metabolism Deoxyglucose--pharmacokinetics Exons Glucose--metabolism Glucose Clamp Technique Glucose Tolerance Test Glucose Transporter Type 4 Insulin--blood Introns Mice Mice, Knockout Monosaccharide Transport Proteins--genetics Muscle Proteins Muscle, Skeletal--drug effects Polymerase Chain Reaction Potassium Channels--deficiency Potassium Channels, Inwardly Rectifying RNA, Messenger--metabolism Receptors, Drug--deficiency Signal Transduction Sodium-Potassium-Exchanging ATPase--metabolism Sulfonylurea Receptors Triglycerides--blood Weight Gain