Wittstein, I S <br/><br/>
Cardiac nitric oxide production due to angiotensin-converting enzyme inhibition decreases beta-adrenergic myocardial contractility in patients with dilated cardiomyopathy.  [electronic resource] 

 -  Journal of the American College of Cardiology  Aug 2001 
 - 429-35 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0735-1097 
<br/><br/><label>Standard No.: </label>10.1016/s0735-1097(01)01404-8  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenergic beta-Antagonists--pharmacology<br/>Angiotensin-Converting Enzyme Inhibitors--pharmacology<br/>Blood Pressure--drug effects<br/>Cardiomyopathy, Dilated--metabolism<br/>Compliance<br/>Depression, Chemical<br/>Diastole<br/>Dobutamine--pharmacology<br/>Enalaprilat--pharmacology<br/>Enzyme Inhibitors--pharmacology<br/>Female<br/>Heart--physiopathology<br/>Hemodynamics--drug effects<br/>Humans<br/>Losartan--pharmacology<br/>Male<br/>Middle Aged<br/>Myocardial Contraction--drug effects<br/>Myocardium--metabolism<br/>Nitric Oxide--biosynthesis<br/>Ventricular Dysfunction, Left--metabolism<br/>omega-N-Methylarginine--pharmacology