Cesselli, D <br/><br/>
Oxidative stress-mediated cardiac cell death is a major determinant of ventricular dysfunction and failure in dog dilated cardiomyopathy.  [electronic resource] 

 -  Circulation research  Aug 2001 
 - 279-86 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1524-4571 
<br/><br/><label>Standard No.: </label>10.1161/hh1501.094115  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing<br/>Adaptor Proteins, Vesicular Transport<br/>Animals<br/>Apoptosis<br/>Blotting, Western<br/>Cardiac Pacing, Artificial<br/>Cardiomyopathy, Dilated--pathology<br/>Caspase 3<br/>Caspase 9<br/>Caspases--metabolism<br/>Cytochrome c Group--metabolism<br/>Disease Models, Animal<br/>Dogs<br/>Enzyme Activation--physiology<br/>Hemodynamics<br/>Immunohistochemistry<br/>In Situ Nick-End Labeling<br/>Myocardium--metabolism<br/>Oxidative Stress<br/>Protein Biosynthesis<br/>Reactive Oxygen Species--metabolism<br/>Shc Signaling Adaptor Proteins<br/>Tumor Suppressor Protein p53--metabolism<br/>Tyrosine--analogs & derivatives<br/>Ventricular Dysfunction--etiology