Clarke, K O <br/><br/>
Tributyrin, an oral butyrate analogue, induces apoptosis through the activation of caspase-3.  [electronic resource] 

 -  Cancer letters  Sep 2001 
 - 57-65 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article 
<br/><br/><label>ISSN: </label>0304-3835 
<br/><br/><label>Standard No.: </label>10.1016/s0304-3835(01)00574-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenocarcinoma--enzymology<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis--drug effects<br/>Butyrates--pharmacology<br/>CDC2-CDC28 Kinases<br/>Caspase 3<br/>Caspases--metabolism<br/>Cell Cycle Proteins<br/>Colonic Neoplasms--enzymology<br/>Cyclin-Dependent Kinase 2<br/>Cyclin-Dependent Kinases--metabolism<br/>DNA-Binding Proteins--metabolism<br/>E2F Transcription Factors<br/>E2F4 Transcription Factor<br/>Enzyme Activation--drug effects<br/>G1 Phase--drug effects<br/>Neoplasm Proteins--metabolism<br/>Nuclear Proteins--metabolism<br/>Phenotype<br/>Phenylbutyrates--pharmacology<br/>Phosphorylation--drug effects<br/>Protein Processing, Post-Translational--drug effects<br/>Protein Serine-Threonine Kinases--metabolism<br/>Retinoblastoma Protein--metabolism<br/>Transcription Factors--metabolism<br/>Triglycerides--pharmacology<br/>Tumor Cells, Cultured--drug effects