Randle, D H <br/><br/>
Differential effects of p19(Arf) and p16(Ink4a) loss on senescence of murine bone marrow-derived preB cells and macrophages.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Aug 2001 
 - 9654-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.171217498  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Apoptosis--radiation effects<br/>B-Lymphocytes--cytology<br/>Bone Marrow Cells--cytology<br/>Cell Line, Transformed<br/>Cell Lineage<br/>Cell Transformation, Viral<br/>Cellular Senescence--physiology<br/>Coculture Techniques<br/>Cyclin-Dependent Kinase Inhibitor p16--deficiency<br/>DNA Methylation<br/>DNA, Complementary--genetics<br/>Exons--genetics<br/>Gene Expression Regulation<br/>Gene Silencing<br/>Genes, p16<br/>Hematopoietic Stem Cells--cytology<br/>Macrophages--cytology<br/>Mice<br/>Promoter Regions, Genetic<br/>Proteins--genetics<br/>Signal Transduction--genetics<br/>Simian virus 40--physiology<br/>Transcription, Genetic<br/>Tumor Suppressor Protein p14ARF