Yoshida, M <br/><br/>
Hmg-CoA reductase inhibitor modulates monocyte-endothelial cell interaction under physiological flow conditions in vitro: involvement of Rho GTPase-dependent mechanism.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  Jul 2001 
 - 1165-71 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/hq0701.092143  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>ADP Ribose Transferases--pharmacology<br/>Actins--metabolism<br/>Botulinum Toxins<br/>Cell Adhesion<br/>Cell Line<br/>Cell Membrane--metabolism<br/>Cells, Cultured<br/>Down-Regulation<br/>Endothelium, Vascular--drug effects<br/>Humans<br/>Hydroxymethylglutaryl-CoA Reductase Inhibitors--pharmacology<br/>Integrins--metabolism<br/>Monocytes--drug effects<br/>Mutation<br/>Polymers--metabolism<br/>Protein Transport<br/>Pyridines--pharmacology<br/>U937 Cells<br/>rhoA GTP-Binding Protein--antagonists & inhibitors