Shuto, T

Activation of NF-kappa B by nontypeable Hemophilus influenzae is mediated by toll-like receptor 2-TAK1-dependent NIK-IKK alpha /beta-I kappa B alpha and MKK3/6-p38 MAP kinase signaling pathways in epithelial cells. [electronic resource] - Proceedings of the National Academy of Sciences of the United States of America Jul 2001 - 8774-9 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0027-8424

10.1073/pnas.151236098 doi


Calcium-Calmodulin-Dependent Protein Kinases--metabolism
DNA-Binding Proteins--metabolism
Drosophila Proteins
Epithelial Cells
Haemophilus influenzae--physiology
HeLa Cells
Humans
I-kappa B Kinase
I-kappa B Proteins
MAP Kinase Kinase 3
MAP Kinase Kinase 6
MAP Kinase Kinase Kinases--metabolism
MAP Kinase Signaling System
Membrane Glycoproteins--metabolism
Mitogen-Activated Protein Kinase Kinases--metabolism
Mitogen-Activated Protein Kinases--metabolism
NF-KappaB Inhibitor alpha
NF-kappa B--metabolism
Phosphorylation
Protein Serine-Threonine Kinases--metabolism
Protein-Tyrosine Kinases--metabolism
Receptors, Cell Surface--metabolism
Toll-Like Receptor 2
Toll-Like Receptors
Tumor Cells, Cultured
p38 Mitogen-Activated Protein Kinases
NF-kappaB-Inducing Kinase