Activation of NF-kappa B by nontypeable Hemophilus influenzae is mediated by toll-like receptor 2-TAK1-dependent NIK-IKK alpha /beta-I kappa B alpha and MKK3/6-p38 MAP kinase signaling pathways in epithelial cells. [electronic resource]
- Proceedings of the National Academy of Sciences of the United States of America Jul 2001
- 8774-9 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0027-8424
10.1073/pnas.151236098 doi
Calcium-Calmodulin-Dependent Protein Kinases--metabolism DNA-Binding Proteins--metabolism Drosophila Proteins Epithelial Cells Haemophilus influenzae--physiology HeLa Cells Humans I-kappa B Kinase I-kappa B Proteins MAP Kinase Kinase 3 MAP Kinase Kinase 6 MAP Kinase Kinase Kinases--metabolism MAP Kinase Signaling System Membrane Glycoproteins--metabolism Mitogen-Activated Protein Kinase Kinases--metabolism Mitogen-Activated Protein Kinases--metabolism NF-KappaB Inhibitor alpha NF-kappa B--metabolism Phosphorylation Protein Serine-Threonine Kinases--metabolism Protein-Tyrosine Kinases--metabolism Receptors, Cell Surface--metabolism Toll-Like Receptor 2 Toll-Like Receptors Tumor Cells, Cultured p38 Mitogen-Activated Protein Kinases NF-kappaB-Inducing Kinase