Fitzjohn, S M <br/><br/>
Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  Jul 2001 
 - 4691-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging--genetics<br/>Alzheimer Disease--genetics<br/>Amyloid beta-Protein Precursor--biosynthesis<br/>Animals<br/>Disease Models, Animal<br/>Electric Stimulation<br/>Excitatory Amino Acid Antagonists--pharmacology<br/>Excitatory Postsynaptic Potentials--drug effects<br/>Genetic Predisposition to Disease<br/>Hippocampus--drug effects<br/>Humans<br/>In Vitro Techniques<br/>Kynurenic Acid--pharmacology<br/>Long-Term Potentiation--genetics<br/>Mice<br/>Mice, Transgenic<br/>Mutation<br/>Neuronal Plasticity<br/>Plaque, Amyloid--pathology<br/>Synaptic Transmission--drug effects