Cheng, J J <br/><br/>
Sequential activation of protein kinase C (PKC)-alpha and PKC-epsilon contributes to sustained Raf/ERK1/2 activation in endothelial cells under mechanical strain.  [electronic resource] 

 -  The Journal of biological chemistry  Aug 2001 
 - 31368-75 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M011317200  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Autoantigens--genetics<br/>Calcium--metabolism<br/>Cattle<br/>DNA-Binding Proteins<br/>Endothelium, Vascular--enzymology<br/>Enzyme Activation<br/>Isoenzymes--physiology<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinase 3<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Oligonucleotides, Antisense--pharmacology<br/>Potassium Channels--genetics<br/>Protein Kinase C--physiology<br/>Protein Kinase C-alpha<br/>Protein Kinase C-epsilon<br/>Proto-Oncogene Proteins<br/>Proto-Oncogene Proteins c-raf--metabolism<br/>Stress, Mechanical<br/>Transcription Factors<br/>Transcription, Genetic<br/>ets-Domain Protein Elk-1