Hübner, C A <br/><br/>
Disruption of KCC2 reveals an essential role of K-Cl cotransport already in early synaptic inhibition.  [electronic resource] 

 -  Neuron  May 2001 
 - 515-24 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0896-6273 
<br/><br/><label>Standard No.: </label>10.1016/s0896-6273(01)00297-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Action Potentials--drug effects<br/>Animals<br/>Animals, Newborn<br/>Carrier Proteins--genetics<br/>Embryo, Mammalian<br/>Embryonic and Fetal Development<br/>Gene Expression Regulation, Developmental<br/>Glycine--pharmacology<br/>Hypoxia--genetics<br/>Mice<br/>Mice, Knockout<br/>Motor Neurons--drug effects<br/>Patch-Clamp Techniques<br/>Potassium--metabolism<br/>Protein Isoforms--deficiency<br/>Sodium--metabolism<br/>Symporters<br/>Synaptic Transmission--drug effects<br/>Tetrodotoxin--pharmacology<br/>gamma-Aminobutyric Acid--pharmacology<br/>K Cl- Cotransporters