Matsuzawa, S I <br/><br/>
Siah-1, SIP, and Ebi collaborate in a novel pathway for beta-catenin degradation linked to p53 responses.  [electronic resource] 

 -  Molecular cell  May 2001 
 - 915-26 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1097-2765 
<br/><br/><label>Standard No.: </label>10.1016/s1097-2765(01)00242-8  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Sequence<br/>Calcium-Binding Proteins<br/>Carrier Proteins--physiology<br/>Cell Cycle--drug effects<br/>Cell Cycle Proteins<br/>Cytoskeletal Proteins--drug effects<br/>DNA Damage--physiology<br/>DNA-Binding Proteins--drug effects<br/>Drosophila Proteins<br/>GTP-Binding Proteins<br/>Heat-Shock Proteins<br/>Humans<br/>Insect Proteins--metabolism<br/>Lymphoid Enhancer-Binding Factor 1<br/>Molecular Sequence Data<br/>Nuclear Proteins--metabolism<br/>Protein Binding<br/>Sequence Alignment<br/>Signal Transduction--drug effects<br/>Trans-Activators<br/>Transcription Factors--drug effects<br/>Tumor Cells, Cultured<br/>Tumor Suppressor Protein p53--pharmacology<br/>Two-Hybrid System Techniques<br/>Ubiquitin-Protein Ligases<br/>beta Catenin<br/>Seven in Absentia Proteins