Jones, N <br/><br/>
Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function.  [electronic resource] 

 -  EMBO reports  May 2001 
 - 438-45 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1469-221X 
<br/><br/><label>Standard No.: </label>10.1093/embo-reports/kve093  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Actins--metabolism<br/>Animals<br/>Apoptosis<br/>Blood Vessels--embryology<br/>Embryonic and Fetal Development<br/>Endothelium, Vascular--cytology<br/>Heart--embryology<br/>Hemorrhage<br/>Immunohistochemistry<br/>In Situ Nick-End Labeling<br/>Mice<br/>Mice, Transgenic<br/>Neovascularization, Physiologic<br/>Receptor Protein-Tyrosine Kinases--genetics<br/>Receptor, TIE-2<br/>Signal Transduction<br/>Transgenes<br/>von Willebrand Factor--metabolism