Lai, J H <br/><br/>
Western and Chinese antirheumatic drug-induced T cell apoptotic DNA damage uses different caspase cascades and is independent of Fas/Fas ligand interaction.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Jun 2001 
 - 6914-24 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.166.11.6914  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alkaloids--toxicity<br/>Anti-Inflammatory Agents, Non-Steroidal--toxicity<br/>Apoptosis--drug effects<br/>Benzylisoquinolines<br/>Caspase 3<br/>Caspase 8<br/>Caspase 9<br/>Caspase Inhibitors<br/>Caspases--biosynthesis<br/>Cell Death--drug effects<br/>Cysteine Proteinase Inhibitors--pharmacology<br/>DNA Damage--immunology<br/>Drugs, Chinese Herbal--toxicity<br/>Enzyme Activation--drug effects<br/>Enzyme Induction--drug effects<br/>Fas Ligand Protein<br/>Humans<br/>Hydroxychloroquine--toxicity<br/>Immunity, Innate--drug effects<br/>Jurkat Cells--cytology<br/>Lymphocyte Activation--drug effects<br/>Lymphocyte Count<br/>Membrane Glycoproteins--biosynthesis<br/>Methotrexate--toxicity<br/>Signal Transduction--drug effects<br/>T-Lymphocyte Subsets--cytology<br/>Tripterygium<br/>U937 Cells<br/>fas Receptor--biosynthesis