Zheng, Y <br/><br/>
NF-kappa B RelA (p65) is essential for TNF-alpha-induced fas expression but dispensable for both TCR-induced expression and activation-induced cell death.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Apr 2001 
 - 4949-57 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.166.8.4949  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Apoptosis--drug effects<br/>Base Sequence<br/>Binding Sites--genetics<br/>CD4-Positive T-Lymphocytes--cytology<br/>Cells, Cultured<br/>Cloning, Molecular<br/>Down-Regulation--genetics<br/>Fas Ligand Protein<br/>Fibroblasts--metabolism<br/>Humans<br/>Jurkat Cells<br/>Ligands<br/>Lymphocyte Activation--drug effects<br/>Membrane Glycoproteins--biosynthesis<br/>Mice<br/>Mice, Inbred C57BL<br/>Molecular Sequence Data<br/>NF-kappa B--deficiency<br/>NF-kappa B p50 Subunit<br/>Phytohemagglutinins--pharmacology<br/>Promoter Regions, Genetic--immunology<br/>Proto-Oncogene Proteins c-rel--deficiency<br/>Receptors, Antigen, T-Cell--physiology<br/>Tetradecanoylphorbol Acetate--pharmacology<br/>Transcription Factor RelA<br/>Tumor Necrosis Factor-alpha--physiology<br/>fas Receptor--biosynthesis