Huang, N K <br/><br/>
Activation of protein kinase A and atypical protein kinase C by A(2A) adenosine receptors antagonizes apoptosis due to serum deprivation in PC12 cells.  [electronic resource] 

 -  The Journal of biological chemistry  Apr 2001 
 - 13838-46 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M008589200  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine--analogs & derivatives<br/>Animals<br/>Apoptosis<br/>Blotting, Western<br/>Cell Membrane--metabolism<br/>Cell Nucleus--metabolism<br/>Cell Survival<br/>Chromones--pharmacology<br/>Colforsin--pharmacology<br/>Coloring Agents--pharmacology<br/>Culture Media, Serum-Free--metabolism<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Cytosol--metabolism<br/>DNA Fragmentation<br/>Dose-Response Relationship, Drug<br/>Down-Regulation<br/>Enzyme Activation<br/>Enzyme Inhibitors--pharmacology<br/>Flow Cytometry<br/>Genes, Dominant<br/>MAP Kinase Signaling System<br/>Models, Biological<br/>Morpholines--pharmacology<br/>PC12 Cells<br/>Phenethylamines--pharmacology<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Phosphoprotein Phosphatases--metabolism<br/>Protein Kinase C--metabolism<br/>Protein Serine-Threonine Kinases<br/>Proto-Oncogene Proteins--genetics<br/>Proto-Oncogene Proteins c-akt<br/>Rats<br/>Receptor, Adenosine A2A<br/>Receptors, Purinergic P1--metabolism<br/>Signal Transduction<br/>Tetrazolium Salts--pharmacology<br/>Thiazoles--pharmacology<br/>Time Factors<br/>Transfection