Beitner-Johnson, D <br/><br/>
Hypoxia activates Akt and induces phosphorylation of GSK-3 in PC12 cells.  [electronic resource] 

 -  Cellular signalling  Jan 2001 
 - 23-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0898-6568 
<br/><br/><label>Standard No.: </label>10.1016/s0898-6568(00)00128-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androstadienes--metabolism<br/>Animals<br/>Calcium-Calmodulin-Dependent Protein Kinases--metabolism<br/>Cell Hypoxia--physiology<br/>Glycogen Synthase Kinase 3<br/>Glycogen Synthase Kinases<br/>PC12 Cells<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphorylation<br/>Phosphotransferases--metabolism<br/>Protein Isoforms<br/>Protein Serine-Threonine Kinases--metabolism<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-akt<br/>Rats<br/>Signal Transduction--physiology<br/>Substrate Specificity<br/>Transcription Factors--drug effects<br/>Wortmannin