Forcet, C <br/><br/>
The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Mar 2001 
 - 3416-21 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.051378298  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis<br/>Caspase 3<br/>Caspase 9<br/>Caspases--metabolism<br/>Cell Adhesion Molecules--genetics<br/>Cell Line, Transformed<br/>Colorectal Neoplasms<br/>DCC Receptor<br/>Enzyme Activation<br/>Genes, Tumor Suppressor<br/>Humans<br/>Receptors, Cell Surface<br/>Tumor Cells, Cultured<br/>Tumor Suppressor Proteins--genetics