Zervas, M <br/><br/>
Neurons in Niemann-Pick disease type C accumulate gangliosides as well as unesterified cholesterol and undergo dendritic and axonal alterations.  [electronic resource] 

 -  Journal of neuropathology and experimental neurology  Jan 2001 
 - 49-64 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-3069 
<br/><br/><label>Standard No.: </label>10.1093/jnen/60.1.49  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antigens, CD--metabolism<br/>Axons--pathology<br/>Cats<br/>Cerebral Cortex--metabolism<br/>Child<br/>Child, Preschool<br/>Cholesterol--metabolism<br/>Dendrites--pathology<br/>Esterification<br/>Filipin--metabolism<br/>G(M2) Ganglioside--metabolism<br/>Gangliosides--metabolism<br/>Genotype<br/>Humans<br/>Intracellular Signaling Peptides and Proteins<br/>Lysosomal Membrane Proteins<br/>Membrane Glycoproteins--metabolism<br/>Mice<br/>Mutation<br/>Neurons--metabolism<br/>Niemann-Pick C1 Protein<br/>Niemann-Pick Diseases--genetics<br/>Parvalbumins--metabolism<br/>Proteins--genetics<br/>Reference Values