Domachowske, J B <br/><br/>
MIP-1alpha is produced but it does not control pulmonary inflammation in response to respiratory syncytial virus infection in mice.  [electronic resource] 

 -  Cellular immunology  Nov 2000 
 - 1-6 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0008-8749 
<br/><br/><label>Standard No.: </label>10.1006/cimm.2000.1730  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acute Disease<br/>Animals<br/>Bronchoalveolar Lavage Fluid--cytology<br/>Chemokine CCL3<br/>Chemokine CCL4<br/>Disease Models, Animal<br/>Gene Expression Regulation, Viral<br/>Humans<br/>Leukocyte Count<br/>Macrophage Inflammatory Proteins--biosynthesis<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Pneumonia, Viral--metabolism<br/>Pneumovirus--physiology<br/>Receptors, CCR1<br/>Receptors, Chemokine--physiology<br/>Respiratory Syncytial Virus Infections--metabolism<br/>Respiratory Syncytial Viruses--physiology<br/>Species Specificity<br/>Specific Pathogen-Free Organisms