Bohlen, H G <br/><br/>
Arteriolar nitric oxide concentration is decreased during hyperglycemia-induced betaII PKC activation.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Feb 2001 
 - H621-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0363-6135 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.2001.280.2.H621  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylcholine--pharmacology<br/>Animals<br/>Arterioles--metabolism<br/>Endothelium, Vascular--enzymology<br/>Enzyme Activation--drug effects<br/>Enzyme Inhibitors--pharmacology<br/>Hyperglycemia--metabolism<br/>Hypertonic Solutions--pharmacology<br/>Indoles--pharmacology<br/>Isoenzymes--metabolism<br/>Jejunum--blood supply<br/>Male<br/>Maleimides--pharmacology<br/>Nitric Oxide--metabolism<br/>Nitroprusside--pharmacology<br/>Osmotic Pressure<br/>Protein Kinase C--metabolism<br/>Protein Kinase C beta<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sodium Chloride--pharmacology<br/>Vasoconstrictor Agents--pharmacology<br/>Vasodilator Agents--pharmacology