Klingbeil, C K <br/><br/>
Targeting Pyk2 to beta 1-integrin-containing focal contacts rescues fibronectin-stimulated signaling and haptotactic motility defects of focal adhesion kinase-null cells.  [electronic resource] 

 -  The Journal of cell biology  Jan 2001 
 - 97-110 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0021-9525 
<br/><br/><label>Standard No.: </label>10.1083/jcb.152.1.97  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Movement--physiology<br/>Cells, Cultured<br/>Cytoskeletal Proteins--metabolism<br/>Enzyme Activation<br/>Fibroblasts--cytology<br/>Fibronectins--metabolism<br/>Focal Adhesion Kinase 1<br/>Focal Adhesion Kinase 2<br/>Focal Adhesion Protein-Tyrosine Kinases<br/>Gene Expression<br/>Humans<br/>Integrin beta1--metabolism<br/>JNK Mitogen-Activated Protein Kinases<br/>Mice<br/>Mice, Knockout<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Paxillin<br/>Phosphoproteins--metabolism<br/>Protein-Tyrosine Kinases--genetics<br/>Recombinant Fusion Proteins--genetics<br/>Signal Transduction