Friedrichsen, B N
Growth hormone- and prolactin-induced proliferation of insulinoma cells, INS-1, depends on activation of STAT5 (signal transducer and activator of transcription 5). [electronic resource]
- Molecular endocrinology (Baltimore, Md.) Jan 2001
- 136-48 p. digital
Publication Type: Journal Article
ISSN: 0888-8809
Standard No.: 10.1210/mend.15.1.0576 doi
Subjects--Topical Terms: Animals Animals, Newborn Cell Division--drug effects Cells, Cultured Cyclic AMP-Dependent Protein Kinases--antagonists & inhibitors Cyclin D2 Cyclins--analysis DNA--biosynthesis DNA-Binding Proteins--genetics Enzyme Inhibitors--pharmacology Gene Expression Human Growth Hormone--pharmacology Humans Insulin--biosynthesis Insulinoma--pathology Islets of Langerhans--cytology MAP Kinase Kinase Kinase 1 Milk Proteins Mitogen-Activated Protein Kinases--antagonists & inhibitors Mutation Pancreatic Neoplasms--pathology Phosphoinositide-3 Kinase Inhibitors Prolactin--pharmacology Protein Kinase C--antagonists & inhibitors Protein Serine-Threonine Kinases--antagonists & inhibitors Protein-Tyrosine Kinases--antagonists & inhibitors RNA, Messenger--analysis Rats Receptors, Prolactin--genetics STAT5 Transcription Factor Signal Transduction Trans-Activators--genetics Transfection Tumor Cells, Cultured p38 Mitogen-Activated Protein Kinases