Cooke, K R <br/><br/>
Hyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Dec 2000 
 - 6612-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.165.11.6612  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antigens, CD--administration & dosage<br/>Bone Marrow Transplantation--adverse effects<br/>Bronchoalveolar Lavage Fluid--cytology<br/>Cell Movement--immunology<br/>Cells, Cultured<br/>Down-Regulation--immunology<br/>Drug Resistance<br/>Female<br/>Humans<br/>Immune Tolerance<br/>Immunity, Innate<br/>Immunoglobulin Fc Fragments--administration & dosage<br/>Intestinal Mucosa--immunology<br/>Isoantigens--immunology<br/>Lipopolysaccharides--antagonists & inhibitors<br/>Lung--immunology<br/>Mice<br/>Mice, Inbred C3H<br/>Mice, Inbred C57BL<br/>Pneumonia--immunology<br/>Receptors, Tumor Necrosis Factor--administration & dosage<br/>Receptors, Tumor Necrosis Factor, Type II<br/>Syndrome<br/>T-Lymphocytes--immunology<br/>Tumor Necrosis Factor-alpha--antagonists & inhibitors