Im, S H <br/><br/>
Mechanism of nasal tolerance induced by a recombinant fragment of acetylcholine receptor for treatment of experimental myasthenia gravis.  [electronic resource] 

 -  Journal of neuroimmunology  Nov 2000 
 - 161-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0165-5728 
<br/><br/><label>Standard No.: </label>10.1016/s0165-5728(00)00395-7  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Administration, Intranasal<br/>Animals<br/>Antigen Presentation--immunology<br/>Female<br/>Immune Tolerance--drug effects<br/>Immunotherapy<br/>Myasthenia Gravis, Autoimmune, Experimental--immunology<br/>Peptide Fragments--immunology<br/>Rats<br/>Rats, Inbred Lew<br/>Receptors, Cholinergic--immunology<br/>Recombinant Proteins--immunology<br/>Th1 Cells--immunology<br/>Th2 Cells--immunology