Sacco, M G <br/><br/>
Lymphoid abnormalities in CD40 ligand transgenic mice suggest the need for tight regulation in gene therapy approaches to hyper immunoglobulin M (IgM) syndrome.  [electronic resource] 

 -  Cancer gene therapy  Oct 2000 
 - 1299-306 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0929-1903 
<br/><br/><label>Standard No.: </label>10.1038/sj.cgt.7700232  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>B-Lymphocytes--immunology<br/>Blotting, Northern<br/>CD40 Antigens--genetics<br/>CD40 Ligand--genetics<br/>Cells, Cultured<br/>DNA Primers--chemistry<br/>Flow Cytometry<br/>Gene Expression<br/>Genetic Therapy--methods<br/>Humans<br/>Hypergammaglobulinemia--immunology<br/>Immunoenzyme Techniques<br/>Immunoglobulin M<br/>Lymph Nodes--ultrastructure<br/>Lymphocyte Activation<br/>Lymphoma, B-Cell--etiology<br/>Mice<br/>Mice, Transgenic<br/>Polymerase Chain Reaction<br/>T-Lymphocytes--immunology