Weskamp, M <br/><br/>
Characterization of the increase in [Ca(2+)](i) during hypotonic shock and the involvement of Ca(2+)-activated K(+) channels in the regulatory volume decrease in human osteoblast-like cells.  [electronic resource] 

 -  The Journal of membrane biology  Nov 2000 
 - 11-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-2631 
<br/><br/><label>Standard No.: </label>10.1007/s002320010010  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Calcium--metabolism<br/>Cell Line<br/>Charybdotoxin--pharmacology<br/>Electrophysiology<br/>Humans<br/>Hypotonic Solutions<br/>Indoles--pharmacology<br/>Intermediate-Conductance Calcium-Activated Potassium Channels<br/>Large-Conductance Calcium-Activated Potassium Channel alpha Subunits<br/>Large-Conductance Calcium-Activated Potassium Channels<br/>Osteoblasts--drug effects<br/>Potassium Channel Blockers<br/>Potassium Channels--physiology<br/>Potassium Channels, Calcium-Activated