Austin Taylor, M <br/><br/>
Functional defects of NK cells treated with chloroquine mimic the lytic defects observed in perforin-deficient mice.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Nov 2000 
 - 5048-53 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.165.9.5048  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bone Marrow Cells--drug effects<br/>Bone Marrow Transplantation--immunology<br/>Cell Movement--drug effects<br/>Cells, Cultured<br/>Chloroquine--pharmacology<br/>Cytotoxicity, Immunologic--drug effects<br/>Fas Ligand Protein<br/>Graft Rejection--genetics<br/>Histocompatibility Antigens Class I--genetics<br/>Humans<br/>Immunosuppressive Agents--pharmacology<br/>Injections, Intravenous<br/>Jurkat Cells<br/>Killer Cells, Natural--drug effects<br/>Ligands<br/>Lung Neoplasms--immunology<br/>Membrane Glycoproteins--antagonists & inhibitors<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred Strains<br/>Mice, Knockout<br/>Perforin<br/>Pore Forming Cytotoxic Proteins<br/>Protein Processing, Post-Translational--drug effects<br/>Tumor Cells, Cultured--drug effects<br/>fas Receptor--metabolism