Hartmann, W <br/><br/>
p57(KIP2) is not mutated in hepatoblastoma but shows increased transcriptional activity in a comparative analysis of the three imprinted genes p57(KIP2), IGF2, and H19.  [electronic resource] 

 -  The American journal of pathology  Oct 2000 
 - 1393-403 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0002-9440 
<br/><br/><label>Standard No.: </label>10.1016/S0002-9440(10)64652-4  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Child, Preschool<br/>Cyclin-Dependent Kinase Inhibitor p57<br/>Female<br/>Genomic Imprinting<br/>Hepatoblastoma--genetics<br/>Humans<br/>Infant<br/>Insulin-Like Growth Factor II--genetics<br/>Liver Neoplasms--genetics<br/>Loss of Heterozygosity<br/>Male<br/>Mutation<br/>Nuclear Proteins--genetics<br/>Polymorphism, Single-Stranded Conformational<br/>RNA, Long Noncoding<br/>RNA, Messenger--metabolism<br/>RNA, Untranslated--genetics<br/>Transcription, Genetic