Grb3-3 is up-regulated in HIV-1-infected T-cells and can potentiate cell activation through NFATc. [electronic resource]
- The Journal of biological chemistry Oct 2000
- 30925-33 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0021-9258
10.1074/jbc.M005535200 doi
Adaptor Proteins, Signal Transducing Adult Antibodies, Monoclonal--metabolism Blotting, Western CD28 Antigens--metabolism CD4-Positive T-Lymphocytes--metabolism Cell Nucleus--metabolism Cyclosporine--pharmacology Cytoplasm--metabolism DNA-Binding Proteins--genetics Female GRB2 Adaptor Protein Gene Products, nef--metabolism Gene Products, tat--metabolism HIV Infections--metabolism HIV-1--metabolism Humans Immunosuppressive Agents--pharmacology Jurkat Cells Leukocytes, Mononuclear--virology Luciferases--metabolism MAP Kinase Kinase Kinase 1 MAP Kinase Signaling System Male Middle Aged NFATC Transcription Factors Nuclear Proteins Plasmids--metabolism Promoter Regions, Genetic Protein Isoforms Protein Serine-Threonine Kinases--metabolism Proteins--chemistry RNA, Messenger--metabolism Receptors, Antigen, T-Cell--metabolism Signal Transduction Terminal Repeat Sequences Time Factors Transcription Factor AP-1--metabolism Transcription Factors--genetics Transfection Up-Regulation nef Gene Products, Human Immunodeficiency Virus tat Gene Products, Human Immunodeficiency Virus