Inoue, K <br/><br/>
Disruption of the ARF transcriptional activator DMP1 facilitates cell immortalization, Ras transformation, and tumorigenesis.  [electronic resource] 

 -  Genes & development  Jul 2000 
 - 1797-809 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0890-9369 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Age Factors<br/>Animals<br/>Cell Division<br/>Cells, Cultured<br/>DNA, Complementary--metabolism<br/>Female<br/>Fibroblasts--metabolism<br/>Gene Library<br/>Genotype<br/>Heterozygote<br/>Humans<br/>Interleukin-2--pharmacology<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Models, Genetic<br/>Mutagenesis, Site-Directed<br/>Neoplasms, Experimental--chemically induced<br/>Phenotype<br/>Proteins--metabolism<br/>T-Lymphocytes--cytology<br/>Time Factors<br/>Tissue Distribution<br/>Transcription Factors--biosynthesis<br/>Transformation, Genetic<br/>Tumor Suppressor Protein p14ARF<br/>Tumor Suppressor Protein p53--metabolism<br/>ras Proteins--metabolism