Ling, Z <br/><br/>
Interleukin-1beta-induced alteration in a beta-cell phenotype can reduce cellular sensitivity to conditions that cause necrosis but not to cytokine-induced apoptosis.  [electronic resource] 

 -  Diabetes  Mar 2000 
 - 340-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0012-1797 
<br/><br/><label>Standard No.: </label>10.2337/diabetes.49.3.340  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alloxan--pharmacology<br/>Animals<br/>Apoptosis--physiology<br/>Cytokines--physiology<br/>Dose-Response Relationship, Drug<br/>Interleukin-1--pharmacology<br/>Islets of Langerhans--drug effects<br/>Male<br/>Necrosis<br/>Nitric Oxide Donors--pharmacology<br/>Osmolar Concentration<br/>Phenotype<br/>Proteins--metabolism<br/>Rats<br/>Rats, Wistar<br/>Streptozocin--pharmacology<br/>Triazoles--pharmacology