Kawahara, M <br/><br/>
Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line.  [electronic resource] 

 -  The Journal of biological chemistry  May 2000 
 - 14077-83 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.275.19.14077  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--metabolism<br/>Amyloid--metabolism<br/>Amyloid beta-Peptides--metabolism<br/>Calcium--metabolism<br/>Cell Line, Transformed<br/>Cholesterol--metabolism<br/>Gonadotropin-Releasing Hormone--metabolism<br/>Homeostasis<br/>Humans<br/>Hypothalamus--cytology<br/>Islet Amyloid Polypeptide<br/>Islets of Langerhans--metabolism<br/>Neurons--metabolism<br/>Prions--metabolism