Kodavanti, U P <br/><br/>
The spontaneously hypertensive rat as a model of human cardiovascular disease: evidence of exacerbated cardiopulmonary injury and oxidative stress from inhaled emission particulate matter.  [electronic resource] 

 -  Toxicology and applied pharmacology  May 2000 
 - 250-63 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0041-008X 
<br/><br/><label>Standard No.: </label>10.1006/taap.2000.8899  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Air Pollutants--toxicity<br/>Animals<br/>Bronchoalveolar Lavage Fluid--chemistry<br/>Carbon--toxicity<br/>Coal Ash<br/>Cytokines--genetics<br/>Disease Models, Animal<br/>Electrocardiography<br/>Erythrocytes<br/>Heart Diseases--etiology<br/>Lung--pathology<br/>Lung Diseases--etiology<br/>Male<br/>Myocardium--pathology<br/>Organ Size<br/>Oxidative Stress<br/>Particulate Matter<br/>RNA, Messenger--analysis<br/>Rats<br/>Rats, Inbred SHR<br/>Rats, Inbred WKY<br/>Respiratory Function Tests<br/>Thiobarbituric Acid Reactive Substances--analysis