Roth, J <br/><br/>
Tumor-derived mutations within the DNA-binding domain of p53 that phenotypically resemble the deletion of the proline-rich domain.  [electronic resource] 

 -  Oncogene  Mar 2000 
 - 1834-42 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1203500  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Binding Sites<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclins--genetics<br/>DNA--metabolism<br/>DNA-Binding Proteins--metabolism<br/>Genes, Tumor Suppressor<br/>Humans<br/>Nuclear Proteins--metabolism<br/>Phenotype<br/>Phosphorylation<br/>Proline--genetics<br/>Promoter Regions, Genetic<br/>Sequence Deletion<br/>Transcriptional Activation<br/>Tumor Cells, Cultured<br/>Tumor Protein p73<br/>Tumor Suppressor Protein p53--genetics<br/>Tumor Suppressor Proteins