Giovanni, A <br/><br/>
E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3.  [electronic resource] 

 -  The Journal of biological chemistry  Apr 2000 
 - 11553-60 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.275.16.11553  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amyloid beta-Peptides--pharmacology<br/>Animals<br/>Apoptosis<br/>Carrier Proteins<br/>Caspase 3<br/>Caspases--metabolism<br/>Cell Cycle Proteins--physiology<br/>Cells, Cultured<br/>Cerebral Cortex--cytology<br/>Coumarins--pharmacology<br/>DNA-Binding Proteins--physiology<br/>E2F Transcription Factors<br/>E2F1 Transcription Factor<br/>Enzyme Activation<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Neurons--drug effects<br/>Oligopeptides--pharmacology<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-bcl-2<br/>Retinoblastoma-Binding Protein 1<br/>Transcription Factor DP1<br/>Transcription Factors--physiology<br/>Tumor Suppressor Protein p53--physiology<br/>bcl-2-Associated X Protein