TY  - GEN
AU  - Park,H L
AU  - Bai,C
AU  - Platt,K A
AU  - Matise,M P
AU  - Beeghly,A
AU  - Hui,C C
AU  - Nakashima,M
AU  - Joyner,A L
TI  - Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutation
SN  - 0950-1991
PY  - 2000///0630
KW  - Abnormalities, Multiple
KW  - Alleles
KW  - Animals
KW  - Binding Sites
KW  - Brain
KW  - embryology
KW  - COS Cells
KW  - DNA
KW  - metabolism
KW  - DNA-Binding Proteins
KW  - Diencephalon
KW  - Embryonic and Fetal Development
KW  - Extremities
KW  - Gene Expression
KW  - Hedgehog Proteins
KW  - Humans
KW  - Kruppel-Like Transcription Factors
KW  - Lung
KW  - Mice
KW  - Mice, Transgenic
KW  - Mutagenesis
KW  - Nerve Tissue Proteins
KW  - Notochord
KW  - Nuclear Proteins
KW  - Oncogene Proteins
KW  - genetics
KW  - Proteins
KW  - Repressor Proteins
KW  - Signal Transduction
KW  - physiology
KW  - Spinal Cord
KW  - Trans-Activators
KW  - Transcription Factors
KW  - Xenopus Proteins
KW  - Zinc Finger Protein GLI1
KW  - Zinc Finger Protein Gli2
KW  - Zinc Finger Protein Gli3
KW  - Zinc Fingers
N1  - Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S
UR  - https://doi.org/10.1242/dev.127.8.1593
ER  -