Hicks, S D <br/><br/>
Hypothermia during reperfusion after asphyxial cardiac arrest improves functional recovery and selectively alters stress-induced protein expression.  [electronic resource] 

 -  Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism  Mar 2000 
 - 520-30 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0271-678X 
<br/><br/><label>Standard No.: </label>10.1097/00004647-200003000-00011  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Asphyxia--complications<br/>Cerebellum--metabolism<br/>HSP70 Heat-Shock Proteins--metabolism<br/>Heart Arrest--etiology<br/>Hippocampus--metabolism<br/>Hypothermia, Induced<br/>Male<br/>Motor Activity<br/>Myocardial Reperfusion Injury--mortality<br/>Neurons--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Time Factors