Vatner, S F <br/><br/>
beta-adrenergic receptor signaling: an acute compensatory adjustment-inappropriate for the chronic stress of heart failure? Insights from Gsalpha overexpression and other genetically engineered animal models.  [electronic resource] 

 -  Circulation research  Mar 2000 
 - 502-6 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.; Review 
<br/><br/><label>ISSN: </label>0009-7330 
<br/><br/><label>Standard No.: </label>10.1161/01.res.86.5.502  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Chronic Disease<br/>Disease Models, Animal<br/>GTP-Binding Protein alpha Subunits, Gs--genetics<br/>Gene Expression<br/>Genetic Engineering<br/>Heart Failure--genetics<br/>Receptors, Adrenergic, beta--physiology<br/>Signal Transduction--physiology<br/>Stress, Physiological--genetics