Haltiwanger, B M <br/><br/>
DNA base excision repair in human malaria parasites is predominantly by a long-patch pathway.  [electronic resource] 

 -  Biochemistry  Feb 2000 
 - 763-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0006-2960 
<br/><br/><label>Standard No.: </label>10.1021/bi9923151  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Binding Sites--genetics<br/>Carbon-Oxygen Lyases--metabolism<br/>Cell-Free System--enzymology<br/>DNA Glycosylases<br/>DNA Repair<br/>DNA, Circular--metabolism<br/>DNA, Protozoan--metabolism<br/>DNA-(Apurinic or Apyrimidinic Site) Lyase<br/>Deoxyribonuclease IV (Phage T4-Induced)<br/>Endodeoxyribonucleases--metabolism<br/>Enzyme Activation<br/>Escherichia coli Proteins<br/>Flap Endonucleases<br/>Humans<br/>Malaria, Falciparum--enzymology<br/>N-Glycosyl Hydrolases--metabolism<br/>Plasmids--metabolism<br/>Plasmodium falciparum--enzymology<br/>Uracil-DNA Glycosidase