Glick, A <br/><br/>
Defects in transforming growth factor-beta signaling cooperate with a Ras oncogene to cause rapid aneuploidy and malignant transformation of mouse keratinocytes.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Dec 1999 
 - 14949-54 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.96.26.14949  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aneuploidy<br/>Animals<br/>Calcium--pharmacology<br/>Carcinoma, Squamous Cell--etiology<br/>Cell Transformation, Neoplastic<br/>Dose-Response Relationship, Drug<br/>Drug Resistance<br/>Genes, ras<br/>Keratinocytes--pathology<br/>Mice<br/>Mice, Mutant Strains<br/>Protein Serine-Threonine Kinases<br/>Receptor, Transforming Growth Factor-beta Type II<br/>Receptors, Transforming Growth Factor beta--genetics<br/>Signal Transduction<br/>Transduction, Genetic<br/>Transforming Growth Factor beta--genetics