Stallmeyer, B <br/><br/>
The function of nitric oxide in wound repair: inhibition of inducible nitric oxide-synthase severely impairs wound reepithelialization.  [electronic resource] 

 -  The Journal of investigative dermatology  Dec 1999 
 - 1090-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-202X 
<br/><br/><label>Standard No.: </label>10.1046/j.1523-1747.1999.00784.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Endothelial Growth Factors--genetics<br/>Enzyme Inhibitors--pharmacology<br/>Epithelium--physiology<br/>Female<br/>Fibroblast Growth Factor 10<br/>Fibroblast Growth Factor 7<br/>Fibroblast Growth Factors<br/>Growth Substances--physiology<br/>Keratinocytes--physiology<br/>Lymphokines--genetics<br/>Lysine--analogs & derivatives<br/>Mice<br/>Mice, Inbred BALB C<br/>Nitric Oxide--physiology<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Nitric Oxide Synthase Type II<br/>Proliferating Cell Nuclear Antigen--analysis<br/>RNA, Messenger--analysis<br/>Vascular Endothelial Growth Factor A<br/>Vascular Endothelial Growth Factors<br/>Wound Healing