Bracamonte, M P <br/><br/>
Activation of soluble guanylate cyclase and potassium channels contribute to relaxations to nitric oxide in smooth muscle derived from canine femoral veins.  [electronic resource] 

 -  Journal of cardiovascular pharmacology  Sep 1999 
 - 407-13 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0160-2446 
<br/><br/><label>Standard No.: </label>10.1097/00005344-199909000-00014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apamin--pharmacology<br/>Cell Membrane Permeability<br/>Cyclic GMP--analogs & derivatives<br/>Dogs<br/>Enzyme Activation<br/>Female<br/>Femoral Vein<br/>Guanylate Cyclase--antagonists & inhibitors<br/>In Vitro Techniques<br/>Male<br/>Muscle Relaxation--drug effects<br/>Muscle, Smooth, Vascular--drug effects<br/>Nitric Oxide--pharmacology<br/>Potassium Channel Blockers<br/>Potassium Channels--metabolism<br/>Tetraethylammonium--pharmacology<br/>Vasoconstrictor Agents--pharmacology