Kalinichenko, V V

Norepinephrine-mediated inhibition of antitumor cytotoxic T lymphocyte generation involves a beta-adrenergic receptor mechanism and decreased TNF-alpha gene expression. [electronic resource] - Journal of immunology (Baltimore, Md. : 1950) Sep 1999 - 2492-9 p. digital

Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.

0022-1767


Animals
Antigens, Differentiation--biosynthesis
CD3 Complex--biosynthesis
CD4-Positive T-Lymphocytes--metabolism
CD8-Positive T-Lymphocytes--metabolism
Cytotoxicity, Immunologic--genetics
Female
Gene Expression Regulation, Neoplastic--drug effects
Intracellular Fluid--drug effects
Lymphocyte Activation--genetics
Lymphocyte Subsets--metabolism
Macrophages--drug effects
Melphalan--toxicity
Mice
Mice, Inbred BALB C
Neoplasm Transplantation
Norepinephrine--pharmacology
Plasmacytoma--chemically induced
Propranolol--pharmacology
RNA, Messenger--biosynthesis
Receptors, Adrenergic, beta--physiology
Spleen--immunology
T-Lymphocytes, Cytotoxic--immunology
Tumor Cells, Cultured
Tumor Necrosis Factor-alpha--antagonists & inhibitors