Kojima, S <br/><br/>
TrkB mutant lacking the amino-terminal half of the extracellular portion acts as a functional brain-derived neurotrophic factor receptor.  [electronic resource] 

 -  Biochimica et biophysica acta  Aug 1999 
 - 104-10 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article 
<br/><br/><label>ISSN: </label>0006-3002 
<br/><br/><label>Standard No.: </label>10.1016/s0005-2736(99)00094-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Binding Sites--genetics<br/>Brain-Derived Neurotrophic Factor--metabolism<br/>COS Cells<br/>In Vitro Techniques<br/>Mice<br/>Peptide Fragments--chemistry<br/>Protein Conformation<br/>Receptor Protein-Tyrosine Kinases--chemistry<br/>Receptor, Ciliary Neurotrophic Factor<br/>Receptors, Nerve Growth Factor--chemistry<br/>Recombinant Proteins--chemistry<br/>Sequence Deletion