Li, S D <br/><br/>
Multiple genes in the left half of the cag pathogenicity island of Helicobacter pylori are required for tyrosine kinase-dependent transcription of interleukin-8 in gastric epithelial cells.  [electronic resource] 

 -  Infection and immunity  Aug 1999 
 - 3893-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0019-9567 
<br/><br/><label>Standard No.: </label>10.1128/IAI.67.8.3893-3899.1999  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine--pharmacology<br/>Animals<br/>Benzoquinones<br/>Cell Line<br/>Cyclic GMP-Dependent Protein Kinases<br/>Gastric Mucosa--metabolism<br/>Genes, Bacterial<br/>Helicobacter pylori--genetics<br/>Interleukin-8--genetics<br/>Lactams, Macrocyclic<br/>Protein Kinases--physiology<br/>Protein-Tyrosine Kinases--physiology<br/>Quinones--pharmacology<br/>Rifabutin--analogs & derivatives<br/>Transcription, Genetic<br/>Tumor Necrosis Factor-alpha--pharmacology