Beltinger, C <br/><br/>
Herpes simplex virus thymidine kinase/ganciclovir-induced apoptosis involves ligand-independent death receptor aggregation and activation of caspases.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Jul 1999 
 - 8699-704 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.96.15.8699  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing<br/>Antiviral Agents--pharmacology<br/>Apoptosis--drug effects<br/>Apoptosis Regulatory Proteins<br/>Brefeldin A--pharmacology<br/>Carrier Proteins--metabolism<br/>Caspases--metabolism<br/>Enzyme Activation<br/>Fas Ligand Protein<br/>Fas-Associated Death Domain Protein<br/>Ganciclovir--pharmacology<br/>Genetic Therapy--methods<br/>Humans<br/>Membrane Glycoproteins--metabolism<br/>Protease Inhibitors--pharmacology<br/>Receptors, Tumor Necrosis Factor--metabolism<br/>Simplexvirus--enzymology<br/>TNF-Related Apoptosis-Inducing Ligand<br/>Thymidine Kinase--metabolism<br/>Transfection<br/>Tumor Cells, Cultured<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Tumor Suppressor Protein p53--metabolism