Flores, I <br/><br/>
Diacylglycerol kinase inhibition prevents IL-2-induced G1 to S transition through a phosphatidylinositol-3 kinase-independent mechanism.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Jul 1999 
 - 708-14 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Cycle--drug effects<br/>Cell Cycle Proteins<br/>Cell Line<br/>Culture Media, Serum-Free<br/>Cyclin D3<br/>Cyclin-Dependent Kinase Inhibitor p27<br/>Cyclins--metabolism<br/>Diacylglycerol Kinase--antagonists & inhibitors<br/>Enzyme Activation--drug effects<br/>Enzyme Inhibitors--pharmacology<br/>G1 Phase--drug effects<br/>Gene Expression Regulation--drug effects<br/>Interleukin-2--physiology<br/>Interleukin-4--antagonists & inhibitors<br/>Mice<br/>Microtubule-Associated Proteins--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Piperidines--pharmacology<br/>Protein Serine-Threonine Kinases<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-akt<br/>Proto-Oncogenes--drug effects<br/>Quinazolines--pharmacology<br/>Quinazolinones<br/>Retinoblastoma Protein--metabolism<br/>S Phase--drug effects<br/>T-Lymphocytes, Cytotoxic--cytology<br/>Tumor Suppressor Proteins